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Di laman Kerosakan gigi:
"Menurut teori hidrodinamik, cecair di dalam tubul dentin dipercayai mekanisme dengan mana reseptor sakit yang dicetuskan dalam pulpa gigi.[4] Sejak dentin ini sklerosis menghalang peredaran mana-mana cecair, kesakitan yang sebaliknya akan berfungsi sebagai amaran bakteria menyerang tidak boleh membangun pada mulanya. Oleh yang demikian, karies gigi boleh maju selama tempoh masa yang panjang tanpa sebarang sensitiviti gigi, membolehkan kerugian yang lebih besar struktur gigi.[perlu rujukan]
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