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In page Amanda Fisher:
"Over the following four years, Fisher showed that the transactivator gene tat was essential for virus replication, that truncation of the 3' open reading frame (ORF) disrupts virus cytopathogenicity, and that sor (now called vif) is required for efficient cell to cell transmission of HIV virus. With clinical collaboration from the Walter Reed National Military Medical Center Fisher showed that patient antisera displayed type-specific neutralising properties and that multiple HIV isolates derived from a single patient were biologically diverse and contained replication-competent as well as replication-incompetent cytopathic variants.[citation needed]
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