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In page Cardiorenal syndrome:
"Unlike markers of heart damage or stress such as troponin, creatine kinase, natriuretic peptides, reliable markers for acute kidney injury are lacking.[citation needed] Recently, research has found several biomarkers that can be used for early detection of acute kidney injury before serious loss of organ function may occur. Several of these biomarkers include neutrophil gelatinase-associated lipocalin (NGAL), N-acetyl-B-D-glucosaminidase (NAG), Cystatin C, and kidney injury molecule-1 (KIM-1) which have been shown to be involved in tubular damage.[1] Other biomarkers that have been shown to be useful include BNP, IL-18, and fatty acid binding protein (FABP).[1] However, there is great variability in the measurement of these biomarkers and their use in diagnosing CRS must be assessed.[2]
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