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In page Ataxia–telangiectasia:
"Differentiation of these disorders is often possible with clinical features and selected laboratory tests. In cases where the distinction is unclear, clinical laboratories can identify genetic abnormalities of ATM, aprataxin, and senataxin, and specialty centers can identify abnormalities of the proteins of potentially responsible genes, such as ATM, MRE11, nibrin, TDP1, aprataxin, and senataxin, as well as other proteins important to ATM function, such as ATR, DNA-PK, and RAD50.[citation needed]
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