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In page Cardiorenal syndrome:

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Cardiorenal syndrome (CRS) pathophysiology involves a complex bidirectional interaction between the heart and kidneys.[citation needed] The underlying mechanisms are broadly categorized into hemodynamic and non-hemodynamic factors. Hemodynamic factors primarily include changes in blood flow, such as reduced cardiac output and elevated central venous or intra-abdominal pressures. Non-hemodynamic factors include neurohormonal activation, oxidative stress, and systemic inflammation. These mechanisms often act synergistically, contributing to the progressive dysfunction of both organs.[1]