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In page Herpes simplex virus:

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These observations suggest that MR in HSV infections involves genetic recombination between damaged viral genomes resulting in the production of viable progeny viruses. HSV-1, upon infecting host cells, induces inflammation and oxidative stress.[6] Thus it appears that the HSV genome may be subjected to oxidative DNA damage during infection, and that MR may enhance viral survival and virulence under these conditions.[citation needed]