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In page Cerebroretinal microangiopathy with calcifications and cysts:

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Typical childhood-onset cerebroretinal microangiopathy with calcifications and cysts is caused by compound heterozygous mutations in the conserved telomere maintenance component 1 (CTC1) gene[1] located in chromosome 17p.31.[2][3] A late-onset phenotype without abnormal eye findings from a CTC1 mutation has been reported.[3]CTC1[1] is a component of the CST complex,[4] which is additionally composed of oligonucleotide/oligosaccharide-binding fold containing 1 (coded by OBFC1,[5] also known as STN1) and telomerase capping complex subunit homolog 1 (coded by TEN1).[6] CST complex is evolutionarily conserved.[4] It binds to single-stranded DNA and associates with a fraction of telomeres, potentially protecting them.[citation needed]