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In page Adams–Oliver syndrome:
"Six AOS genes have been identified: ARHGAP31,[2] DOCK6,[3] RBPJ,[4] EOGT,[5][6] NOTCH1,[7][8] and DLL4.[9] ARHGAP31 and DOCK6 are both regulatory proteins that control members of the Rho family of GTPases and specifically regulate the activity of Cdc42 and Rac1. Autosomal dominant mutations in ARHGAP31 (a GTPase-activating protein) and autosomal recessive mutations in DOCK6 (a guanine nucleotide exchange factor) cause an accumulation of the inactive GTPase and lead to defects of the cytoskeleton.[citation needed]
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