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In page Congenital adrenal hyperplasia due to 17α-hydroxylase deficiency:

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Milder forms of this deficiency in genetic females allow some degree of sexual development, with variable reproductive system dysregulation that can include incomplete Tanner scale development, retrograde sexual development, irregular menstruation, early menopause, or – in very mild cases – no physical symptoms beyond infertility.[1][2][3] Evidence suggests that only 5% of normal enzyme activity may be enough to allow at least the physical changes of female puberty, if not ovulation and fertility.[citation needed] In women with mild cases, elevated blood pressure and/or infertility is the presenting clinical problem.